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Fish intake and risk of heart failure: A meta-analysis of five prospective cohort studies.

Experimental and therapeutic medicine

Hou LN, Li F, Zhou Y, Nie SH, Su L, Chen PA, Tan WL, Xu DL.
PMID: 23181122
Exp Ther Med. 2012 Sep;4(3):481-486. doi: 10.3892/etm.2012.605. Epub 2012 Jun 08.

The findings on the association between fish intake and the risk of heart failure (HF) have been inconsistent. The purpose of this study was to clarify this potential association. We searched for relevant studies in the PubMed database through...

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Hou LN, Li F, Zhou Y, et al. Fish intake and risk of heart failure: A meta-analysis of five prospective cohort studies. Exp Ther Med. 2012;4(3):481-486doi: 10.3892/etm.2012.605.
Hou, L. N., Li, F., Zhou, Y., Nie, S. H., Su, L., Chen, P. A., Tan, W. L., & Xu, D. L. (2012). Fish intake and risk of heart failure: A meta-analysis of five prospective cohort studies. Experimental and therapeutic medicine, 4(3), 481-486. https://doi.org/10.3892/etm.2012.605
Hou, Li-Na, et al. "Fish intake and risk of heart failure: A meta-analysis of five prospective cohort studies." Experimental and therapeutic medicine vol. 4,3 (2012): 481-486. doi: https://doi.org/10.3892/etm.2012.605
Hou LN, Li F, Zhou Y, Nie SH, Su L, Chen PA, Tan WL, Xu DL. Fish intake and risk of heart failure: A meta-analysis of five prospective cohort studies. Exp Ther Med. 2012 Sep;4(3):481-486. doi: 10.3892/etm.2012.605. Epub 2012 Jun 08. PMID: 23181122; PMCID: PMC3503629.
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Association of FAS gene polymorphisms with systemic lupus erythematosus: A case-control study and meta-analysis.

Experimental and therapeutic medicine

Lu MM, Ye QL, Feng CC, Yang J, Zhang T, Li J, Leng RX, Pan HF, Yuan H, Ye DQ.
PMID: 23181125
Exp Ther Med. 2012 Sep;4(3):497-502. doi: 10.3892/etm.2012.625. Epub 2012 Jun 28.

The association of functional polymorphisms in the promoter of the apoptosis gene FAS with systemic lupus erythematosus (SLE) susceptibility has been a controversial subject. We conducted a case-control study to investigate this association in a Chinese population and performed...

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Lu MM, Ye QL, Feng CC, et al. Association of FAS gene polymorphisms with systemic lupus erythematosus: A case-control study and meta-analysis. Exp Ther Med. 2012;4(3):497-502doi: 10.3892/etm.2012.625.
Lu, M. M., Ye, Q. L., Feng, C. C., Yang, J., Zhang, T., Li, J., Leng, R. X., Pan, H. F., Yuan, H., & Ye, D. Q. (2012). Association of FAS gene polymorphisms with systemic lupus erythematosus: A case-control study and meta-analysis. Experimental and therapeutic medicine, 4(3), 497-502. https://doi.org/10.3892/etm.2012.625
Lu, Man-Man, et al. "Association of FAS gene polymorphisms with systemic lupus erythematosus: A case-control study and meta-analysis." Experimental and therapeutic medicine vol. 4,3 (2012): 497-502. doi: https://doi.org/10.3892/etm.2012.625
Lu MM, Ye QL, Feng CC, Yang J, Zhang T, Li J, Leng RX, Pan HF, Yuan H, Ye DQ. Association of FAS gene polymorphisms with systemic lupus erythematosus: A case-control study and meta-analysis. Exp Ther Med. 2012 Sep;4(3):497-502. doi: 10.3892/etm.2012.625. Epub 2012 Jun 28. PMID: 23181125; PMCID: PMC3503889.
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Expression levels of multidrug resistance-associated protein 4 (MRP4) in human leukemia and lymphoma cell lines, and the inhibitory effects of the MRP-specific inhibitor MK-571 on methotrexate distribution in rats.

Experimental and therapeutic medicine

Takeuchi K, Shibata M, Kashiyama E, Umehara K.
PMID: 23181130
Exp Ther Med. 2012 Sep;4(3):524-532. doi: 10.3892/etm.2012.627. Epub 2012 Jun 29.

In the development of anti-blood cancer drugs, the chronic myelocytic leukemia (KU812), acute myelocytic leukemia (KG-1) and lymphoma (U937) cell lines are commonly used in preclinical pharmacology studies as human cancer xenograft models in mice. In the present study,...

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Takeuchi K, Shibata M, Kashiyama E, et al. Expression levels of multidrug resistance-associated protein 4 (MRP4) in human leukemia and lymphoma cell lines, and the inhibitory effects of the MRP-specific inhibitor MK-571 on methotrexate distribution in rats. Exp Ther Med. 2012;4(3):524-532doi: 10.3892/etm.2012.627.
Takeuchi, K., Shibata, M., Kashiyama, E., & Umehara, K. (2012). Expression levels of multidrug resistance-associated protein 4 (MRP4) in human leukemia and lymphoma cell lines, and the inhibitory effects of the MRP-specific inhibitor MK-571 on methotrexate distribution in rats. Experimental and therapeutic medicine, 4(3), 524-532. https://doi.org/10.3892/etm.2012.627
Takeuchi, Kenji, et al. "Expression levels of multidrug resistance-associated protein 4 (MRP4) in human leukemia and lymphoma cell lines, and the inhibitory effects of the MRP-specific inhibitor MK-571 on methotrexate distribution in rats." Experimental and therapeutic medicine vol. 4,3 (2012): 524-532. doi: https://doi.org/10.3892/etm.2012.627
Takeuchi K, Shibata M, Kashiyama E, Umehara K. Expression levels of multidrug resistance-associated protein 4 (MRP4) in human leukemia and lymphoma cell lines, and the inhibitory effects of the MRP-specific inhibitor MK-571 on methotrexate distribution in rats. Exp Ther Med. 2012 Sep;4(3):524-532. doi: 10.3892/etm.2012.627. Epub 2012 Jun 29. PMID: 23181130; PMCID: PMC3503844.
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TLR4 antagonist suppresses airway remodeling in asthma by inhibiting the T-helper 2 response.

Experimental and therapeutic medicine

Li M, Wang ZN, Yang LF, Yan Y, Cai LM, Li YT, Qiao YK, Chen ZG.
PMID: 28966674
Exp Ther Med. 2017 Oct;14(4):2911-2916. doi: 10.3892/etm.2017.4898. Epub 2017 Aug 07.

Airway remodeling is a hallmark of bronchial asthma. Our group has previously reported that the thymic stromal lymphopoietin (TSLP), an airway epithelial-derived cytokine, has a central role in the pathogenesis of airway remodeling, and that toll-like receptor (TLR) 4...

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Li M, Wang ZN, Yang LF, et al. TLR4 antagonist suppresses airway remodeling in asthma by inhibiting the T-helper 2 response. Exp Ther Med. 2017;14(4):2911-2916doi: 10.3892/etm.2017.4898.
Li, M., Wang, Z. N., Yang, L. F., Yan, Y., Cai, L. M., Li, Y. T., Qiao, Y. K., & Chen, Z. G. (2017). TLR4 antagonist suppresses airway remodeling in asthma by inhibiting the T-helper 2 response. Experimental and therapeutic medicine, 14(4), 2911-2916. https://doi.org/10.3892/etm.2017.4898
Li, Ming, et al. "TLR4 antagonist suppresses airway remodeling in asthma by inhibiting the T-helper 2 response." Experimental and therapeutic medicine vol. 14,4 (2017): 2911-2916. doi: https://doi.org/10.3892/etm.2017.4898
Li M, Wang ZN, Yang LF, Yan Y, Cai LM, Li YT, Qiao YK, Chen ZG. TLR4 antagonist suppresses airway remodeling in asthma by inhibiting the T-helper 2 response. Exp Ther Med. 2017 Oct;14(4):2911-2916. doi: 10.3892/etm.2017.4898. Epub 2017 Aug 07. PMID: 28966674; PMCID: PMC5613182.
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MicroRNA-186 regulates the invasion and metastasis of bladder cancer via vascular endothelial growth factor C.

Experimental and therapeutic medicine

He X, Ping J, Wen D.
PMID: 28966690
Exp Ther Med. 2017 Oct;14(4):3253-3258. doi: 10.3892/etm.2017.4908. Epub 2017 Aug 08.

The present study aimed to investigate the expression of microRNA (miRNA or miR)-186 in tumor tissue, blood and urine from patients with bladder cancer. The mechanism by which miR-186 regulates the invasion and metastasis of bladder cancer was also...

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He X, Ping J, Wen D. MicroRNA-186 regulates the invasion and metastasis of bladder cancer via vascular endothelial growth factor C. Exp Ther Med. 2017;14(4):3253-3258doi: 10.3892/etm.2017.4908.
He, X., Ping, J., & Wen, D. (2017). MicroRNA-186 regulates the invasion and metastasis of bladder cancer via vascular endothelial growth factor C. Experimental and therapeutic medicine, 14(4), 3253-3258. https://doi.org/10.3892/etm.2017.4908
He, Xuefeng, et al. "MicroRNA-186 regulates the invasion and metastasis of bladder cancer via vascular endothelial growth factor C." Experimental and therapeutic medicine vol. 14,4 (2017): 3253-3258. doi: https://doi.org/10.3892/etm.2017.4908
He X, Ping J, Wen D. MicroRNA-186 regulates the invasion and metastasis of bladder cancer via vascular endothelial growth factor C. Exp Ther Med. 2017 Oct;14(4):3253-3258. doi: 10.3892/etm.2017.4908. Epub 2017 Aug 08. PMID: 28966690; PMCID: PMC5613196.
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Wogonoside alleviates inflammation induced by traumatic spinal cord injury by suppressing NF-κB and NLRP3 inflammasome activation.

Experimental and therapeutic medicine

Zhu Y, Zhu H, Wang Z, Gao F, Wang J, Zhang W.
PMID: 28966691
Exp Ther Med. 2017 Oct;14(4):3304-3308. doi: 10.3892/etm.2017.4904. Epub 2017 Aug 08.

Wogonoside possesses anti-oxidative, anti-inflammatory, anti-allergy and anti-tumor properties. The aim of the present study was to evaluate whether wogonoside alleviates spinal cord injury (SCI)-induced inflammation via nuclear factor (NF)-κB and nucleotide-binding oligomerization domain-like receptor family pyrin domain containing 3...

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Zhu Y, Zhu H, Wang Z, et al. Wogonoside alleviates inflammation induced by traumatic spinal cord injury by suppressing NF-κB and NLRP3 inflammasome activation. Exp Ther Med. 2017;14(4):3304-3308doi: 10.3892/etm.2017.4904.
Zhu, Y., Zhu, H., Wang, Z., Gao, F., Wang, J., & Zhang, W. (2017). Wogonoside alleviates inflammation induced by traumatic spinal cord injury by suppressing NF-κB and NLRP3 inflammasome activation. Experimental and therapeutic medicine, 14(4), 3304-3308. https://doi.org/10.3892/etm.2017.4904
Zhu, Yonglin, et al. "Wogonoside alleviates inflammation induced by traumatic spinal cord injury by suppressing NF-κB and NLRP3 inflammasome activation." Experimental and therapeutic medicine vol. 14,4 (2017): 3304-3308. doi: https://doi.org/10.3892/etm.2017.4904
Zhu Y, Zhu H, Wang Z, Gao F, Wang J, Zhang W. Wogonoside alleviates inflammation induced by traumatic spinal cord injury by suppressing NF-κB and NLRP3 inflammasome activation. Exp Ther Med. 2017 Oct;14(4):3304-3308. doi: 10.3892/etm.2017.4904. Epub 2017 Aug 08. PMID: 28966691; PMCID: PMC5613185.
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Inhibitory effect of burdock leaves on elastase and tyrosinase activity.

Experimental and therapeutic medicine

Horng CT, Wu HC, Chiang NN, Lee CF, Huang YS, Wang HY, Yang JS, Chen FA.
PMID: 28912875
Exp Ther Med. 2017 Oct;14(4):3247-3252. doi: 10.3892/etm.2017.4880. Epub 2017 Aug 03.

Burdock (

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Horng CT, Wu HC, Chiang NN, et al. Inhibitory effect of burdock leaves on elastase and tyrosinase activity. Exp Ther Med. 2017;14(4):3247-3252doi: 10.3892/etm.2017.4880.
Horng, C. T., Wu, H. C., Chiang, N. N., Lee, C. F., Huang, Y. S., Wang, H. Y., Yang, J. S., & Chen, F. A. (2017). Inhibitory effect of burdock leaves on elastase and tyrosinase activity. Experimental and therapeutic medicine, 14(4), 3247-3252. https://doi.org/10.3892/etm.2017.4880
Horng, Chi-Ting, et al. "Inhibitory effect of burdock leaves on elastase and tyrosinase activity." Experimental and therapeutic medicine vol. 14,4 (2017): 3247-3252. doi: https://doi.org/10.3892/etm.2017.4880
Horng CT, Wu HC, Chiang NN, Lee CF, Huang YS, Wang HY, Yang JS, Chen FA. Inhibitory effect of burdock leaves on elastase and tyrosinase activity. Exp Ther Med. 2017 Oct;14(4):3247-3252. doi: 10.3892/etm.2017.4880. Epub 2017 Aug 03. PMID: 28912875; PMCID: PMC5585880.
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A crossover study of the combination therapy of metformin and exenatide or biphasic insulin aspart 30 in overweight or obese patients newly diagnosed with type 2 diabetes mellitus.

Experimental and therapeutic medicine

Quan H, Zhang H, Wei W, Fang T, Chen D, Chen K.
PMID: 28912879
Exp Ther Med. 2017 Oct;14(4):3279-3287. doi: 10.3892/etm.2017.4863. Epub 2017 Aug 02.

The aim of the present study was to explore the effects of various combinations of exenatide, metformin (MET) and biphasic insulin aspart 30 (BIA30) on type 2 diabetes mellitus (T2DM). Two hundred overweight or obese patients newly diagnosed with...

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Quan H, Zhang H, Wei W, et al. A crossover study of the combination therapy of metformin and exenatide or biphasic insulin aspart 30 in overweight or obese patients newly diagnosed with type 2 diabetes mellitus. Exp Ther Med. 2017;14(4):3279-3287doi: 10.3892/etm.2017.4863.
Quan, H., Zhang, H., Wei, W., Fang, T., Chen, D., & Chen, K. (2017). A crossover study of the combination therapy of metformin and exenatide or biphasic insulin aspart 30 in overweight or obese patients newly diagnosed with type 2 diabetes mellitus. Experimental and therapeutic medicine, 14(4), 3279-3287. https://doi.org/10.3892/etm.2017.4863
Quan, Huibiao, et al. "A crossover study of the combination therapy of metformin and exenatide or biphasic insulin aspart 30 in overweight or obese patients newly diagnosed with type 2 diabetes mellitus." Experimental and therapeutic medicine vol. 14,4 (2017): 3279-3287. doi: https://doi.org/10.3892/etm.2017.4863
Quan H, Zhang H, Wei W, Fang T, Chen D, Chen K. A crossover study of the combination therapy of metformin and exenatide or biphasic insulin aspart 30 in overweight or obese patients newly diagnosed with type 2 diabetes mellitus. Exp Ther Med. 2017 Oct;14(4):3279-3287. doi: 10.3892/etm.2017.4863. Epub 2017 Aug 02. PMID: 28912879; PMCID: PMC5585756.
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Immune regulation of miR-30 on the .

Experimental and therapeutic medicine

Wu Y, Sun Q, Dai L.
PMID: 28912881
Exp Ther Med. 2017 Oct;14(4):3299-3303. doi: 10.3892/etm.2017.4872. Epub 2017 Aug 02.

The present study aimed to examine the expression of microRNA (miR)-30 family members in THP-1 human monocytes cells during

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Wu Y, Sun Q, Dai L. Immune regulation of miR-30 on the . Exp Ther Med. 2017;14(4):3299-3303doi: 10.3892/etm.2017.4872.
Wu, Y., Sun, Q., & Dai, L. (2017). Immune regulation of miR-30 on the . Experimental and therapeutic medicine, 14(4), 3299-3303. https://doi.org/10.3892/etm.2017.4872
Wu, Yuqing, et al. "Immune regulation of miR-30 on the ." Experimental and therapeutic medicine vol. 14,4 (2017): 3299-3303. doi: https://doi.org/10.3892/etm.2017.4872
Wu Y, Sun Q, Dai L. Immune regulation of miR-30 on the . Exp Ther Med. 2017 Oct;14(4):3299-3303. doi: 10.3892/etm.2017.4872. Epub 2017 Aug 02. PMID: 28912881; PMCID: PMC5585754.
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Tirofiban facilitates the reperfusion process during endovascular thrombectomy in ICAS.

Experimental and therapeutic medicine

Zhao H, Zhang J, Gu D, Shi Z, Pan J, Geng Y, Shi T.
PMID: 28912883
Exp Ther Med. 2017 Oct;14(4):3314-3318. doi: 10.3892/etm.2017.4856. Epub 2017 Jul 31.

The aim of the present study was to assess the use of tirofiban injections for rescue therapy following artery reocclusion due to intra-luminal thrombosis during endovascular thrombectomy in patients with acute ischemic stroke (AIS). A total of seven cases...

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Zhao H, Zhang J, Gu D, et al. Tirofiban facilitates the reperfusion process during endovascular thrombectomy in ICAS. Exp Ther Med. 2017;14(4):3314-3318doi: 10.3892/etm.2017.4856.
Zhao, H., Zhang, J., Gu, D., Shi, Z., Pan, J., Geng, Y., & Shi, T. (2017). Tirofiban facilitates the reperfusion process during endovascular thrombectomy in ICAS. Experimental and therapeutic medicine, 14(4), 3314-3318. https://doi.org/10.3892/etm.2017.4856
Zhao, Hongchen, et al. "Tirofiban facilitates the reperfusion process during endovascular thrombectomy in ICAS." Experimental and therapeutic medicine vol. 14,4 (2017): 3314-3318. doi: https://doi.org/10.3892/etm.2017.4856
Zhao H, Zhang J, Gu D, Shi Z, Pan J, Geng Y, Shi T. Tirofiban facilitates the reperfusion process during endovascular thrombectomy in ICAS. Exp Ther Med. 2017 Oct;14(4):3314-3318. doi: 10.3892/etm.2017.4856. Epub 2017 Jul 31. PMID: 28912883; PMCID: PMC5585762.
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Role of stem cell growth factor/c-Kit in the pathogenesis of irritable bowel syndrome.

Experimental and therapeutic medicine

Chai Y, Huang Y, Tang H, Tu X, He J, Wang T, Zhang Q, Xiong F, Li D, Qiu Z.
PMID: 28413456
Exp Ther Med. 2017 Apr;13(4):1187-1193. doi: 10.3892/etm.2017.4133. Epub 2017 Feb 20.

Irritable bowel syndrome (IBS) is a functional bowel disease with a complicated etiopathogenesis, often characterized by gastrointestinal motility disorder and high visceral sensitivity. IBS is a comprehensive multi-systemic disorder, with the interaction of multiple factors, such as mental stress,...

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Chai Y, Huang Y, Tang H, et al. Role of stem cell growth factor/c-Kit in the pathogenesis of irritable bowel syndrome. Exp Ther Med. 2017;13(4):1187-1193doi: 10.3892/etm.2017.4133.
Chai, Y., Huang, Y., Tang, H., Tu, X., He, J., Wang, T., Zhang, Q., Xiong, F., Li, D., & Qiu, Z. (2017). Role of stem cell growth factor/c-Kit in the pathogenesis of irritable bowel syndrome. Experimental and therapeutic medicine, 13(4), 1187-1193. https://doi.org/10.3892/etm.2017.4133
Chai, Yuna, et al. "Role of stem cell growth factor/c-Kit in the pathogenesis of irritable bowel syndrome." Experimental and therapeutic medicine vol. 13,4 (2017): 1187-1193. doi: https://doi.org/10.3892/etm.2017.4133
Chai Y, Huang Y, Tang H, Tu X, He J, Wang T, Zhang Q, Xiong F, Li D, Qiu Z. Role of stem cell growth factor/c-Kit in the pathogenesis of irritable bowel syndrome. Exp Ther Med. 2017 Apr;13(4):1187-1193. doi: 10.3892/etm.2017.4133. Epub 2017 Feb 20. PMID: 28413456; PMCID: PMC5377426.
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Persicarin isolated from .

Experimental and therapeutic medicine

Lee JY, Kim MY, Shin SH, Shin MR, Kwon OJ, Kim TH, Park CH, Noh JS, Rhee MH, Roh SS.
PMID: 28413457
Exp Ther Med. 2017 Apr;13(4):1194-1202. doi: 10.3892/etm.2017.4113. Epub 2017 Feb 10.

Persicarin is one of the major components of the

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Lee JY, Kim MY, Shin SH, et al. Persicarin isolated from . Exp Ther Med. 2017;13(4):1194-1202doi: 10.3892/etm.2017.4113.
Lee, J. Y., Kim, M. Y., Shin, S. H., Shin, M. R., Kwon, O. J., Kim, T. H., Park, C. H., Noh, J. S., Rhee, M. H., & Roh, S. S. (2017). Persicarin isolated from . Experimental and therapeutic medicine, 13(4), 1194-1202. https://doi.org/10.3892/etm.2017.4113
Lee, Joo Young, et al. "Persicarin isolated from ." Experimental and therapeutic medicine vol. 13,4 (2017): 1194-1202. doi: https://doi.org/10.3892/etm.2017.4113
Lee JY, Kim MY, Shin SH, Shin MR, Kwon OJ, Kim TH, Park CH, Noh JS, Rhee MH, Roh SS. Persicarin isolated from . Exp Ther Med. 2017 Apr;13(4):1194-1202. doi: 10.3892/etm.2017.4113. Epub 2017 Feb 10. PMID: 28413457; PMCID: PMC5377288.
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Showing 1 to 12 of 11071 entries